Oral Pathology Mnemonics Online Course

BY
Udemy

Mode

Online

Fees

₹ 799

Quick Facts

particular details
Medium of instructions English
Mode of learning Self study
Mode of Delivery Video and Text Based

Course and certificate fees

Fees information
₹ 799
certificate availability

Yes

certificate providing authority

Udemy

The syllabus

Developmental Defects of the tongue

  • Microglossia
  • Macroglossia - introduction
  • Macroglossia- part 2 (Bonus: Cretenism is Congenital Hypohyroidism)
  • Macroglossia - part 3
  • Macroglossia - part 4 (Bonus: Acromegaly and Gigantism)
  • Lingual Thyroid Nodule
  • Quiz for Section 1

Developmental Defects of the Jaws

  • Micrognathia
  • Macrognathia
  • Hemi-facial Hypertrophy
  • Hemi-facial Atrophy
  • Craniofacial Dyostosis (Crouzen's Syndrome)
  • Cleido-Cranial Dysplasia (CCD)
  • Quiz for Section 2

Developmental Alterations in The Number of Teeth

  • Supernumerary Teeth
  • Quiz for Section 3

Developmental Alterations in The Shape of Teeth

  • Gemination
  • Concrescence
  • Dens Invaginatus (Dens in Dente)
  • Enamel Pearls
  • Cervical Enamel Extension

Developmental Alterations in The Structure of Teeth

  • Erosion
  • Hypoplastic Amelogenesis Imperfecta
  • Hypomaturation Amelogenesis Imperfecta
  • Hypomaturation and Hypoplastic Amelogenesis Imperfecta
  • Genes Associated with Ameologenesis Imperfecta
  • Dentin Dysplasia
  • Dentinogenesis Imperfecta

Environmental Alterations of Teeth

  • Turner's Hypoplasia
  • Intrinsic Staining- Ciprofloxacin

Inflammatory Diseases of Bone

  • Alveolar Osteitis (Dry Socket)
  • Focal Sclerosing (condensing) Osteitis
  • Acute Suppurative Osteomyelitis
  • Diffuse Sclerosing Osteomyelitis
  • Chronic Osteomyelitis with Proliferative Periostitis
  • Osteochemonecrosis - Bisphosphonates
  • Bonus: Osteomyelitis

Metabolic Bone Conditions

  • Paget's Disease of Bone
  • Central Giant Cell Granuloma
  • Osteopetrosis
  • Osteogenesis Imperfecta

Benign Fibro-osseous Lesions

  • Periapical Cemento-osseous Dysplasia
  • Florid Cemento-osseous Dysplasia
  • Fibrous Dysplasia
  • Monostotic Fibrous Dysplasia of the Jaws
  • Cherubism

Non-odontogenic Tumors of Bone

  • Osteoma
  • Cemento-Ossifying Fibroma
  • Hemangioma of Bone
  • Osteosarcoma
  • Chondroma and Chondrosarcoma
  • Multiple Myeloma
  • Bonus: Classification of Non-odontogenic Tumors of Bone

Diseases of Bone

  • Langerhans Cell Histiocytosis - part 1
  • Langerhans Cell Histiocytosis - part 2
  • Ewing's Sarcoma
  • Metastatic Tumors in the Jaws

Cysts of the Oral Region

  • Histiogenic Classification of Odontogenic Cysts

Odontogenic Cysts

  • Radicular Cyst
  • Eruption Cyst
  • Dentigerous Cyst + Differences between Dentigerous Cyst and Eruption Cyst
  • Odontogenic Keratocyst (OKC)
  • Lateral Periodontal Cyst
  • Glandular Odontogenic Cyst

Non-epithelial Primary Bone Cyst (pseudocysts)

  • Solitary Bone Cyst
  • Aneurysmal Bone Cyst (ABC)

Odontogenic Tumors

  • Ameloblastoma - part 1
  • Ameloblastoma - part 2
  • Adenomatoid Odontogenic Tumor (AOT)
  • Calcifying Epithelial Odontogenic Tumor (CEOT)
  • Calcifying Odontogenic Cyst/Tumor
  • Squamous Odontogenic Tumor
  • Ameloblastic Fibroma
  • Odontogenic Myxoma

Verrucal & Papillary Lesions

  • Squamous Papilloma
  • Condylomata lata
  • Focal Epithelial Hyperplasia
  • Pyostomatitis Vegetans
  • Verruciform Xanthoma
  • Verruca Vulgaris

Reactive Hyperplastic Fibrous Tissue Lesions

  • Giant Cell Fibroma
  • Epulis Fissuratum
  • Pyogenic Granuloma
  • Peripheral Ossifying Fibroma

Neoplastic Fibrous Tissue Lesions

  • Fibrous Histiocytoma
  • Fibromatosis

Adipose Tissue Lesions

  • Lipoma

Neural Tissue Lesions

  • Neurilemoma
  • Neurofibroma
  • Neurofibromatosis Type I (von Recklinghausen’s Disease)
  • Multiple Endocrine Neoplasia (MEN)
  • Melanotic Neuroectodermal Tumor of Infancy

Vascular Lesions

  • Cavernous Lymphangioma

Soft Tissue Sarcoma

  • Fibrosarcoma
  • Liposarcoma
  • Angiosarcoma
  • Kaposi's Sarcoma

Reactive Lymphoid Lesions

  • Angiolymphoid Hyperplasia with Eosinophilia (ALHE)

Neoplastic Lymphoid Lesions

  • Non-Hodgkin’s Lymphoma
  • Burkitt's Lymphoma
  • Hodgkin's Lymphoma
  • Granulocytic Sarcoma (Extra Medullary Myeloid Tumor)
  • Quiz for Section 24

Benign Salivary Gland Tumors

  • Origins of Benign Salivary gland Tumors
  • Pleomorphic Adenoma
  • Warthin's Tumors
  • Quiz for Section 25

Oral Pigmented lesions -Syndrome Associated Pigmentation

  • Peutz-Jeghers Syndrome
  • McCune-Albright Syndrome
  • Drug-induced Pigmentation

Oral Pigmented lesions- Increased Number of Melanocytes

  • Oral Malignant Melanoma

Oral Pigmented Lesions - Red and Blue Lesions

  • Sturge-Weber Syndrome
  • Hereditary Hemorrhagic Telangectasia

Hereditary White Lesions

  • Leukoedema
  • White Sponge Nevus

Oral Hairy Leukoplakia |Infectious White Lesions

  • Oral Hairy Leukplakia is associated with Epstein-Barr virus
  • Oral Hairy Leukoplakia site: tongue-bilaterally
  • Oral Hairy Leukoplakia: Histology: Presence of koilocyte-like cells

Oral Lichen Planus | Immune Mediated White Lesions

  • Striated OLP presents with hyperkeratosis and hyperparakeratosis
  • OLP Histology: Band-like lymphocyte infiltration (mainly CD8 Cells)
  • OLP Histology: Civatte bodies, Basal Cell Layer Degeneration, Vaculative necrosi
  • OLP Histology: Saw-tooth Rete Ridges in Skin
  • OLP is associated with hepatitis C
  • OLP- Civatte Story wrap-up
  • OLP Occurs most commonly in older females
  • OLP has 4 clinical presentations
  • OLP- Wickham's Striae (skin lesions)

Discoid Lupus Erythematosus | Immune Mediated White Lesions

  • DLE is most common in middle-aged females.
  • DLE: Skin lesions on face and scalp
  • DLE causes hair loss
  • DLE: the oral sites most commonly affected
  • DLE: Oral clinical presentation
  • SLE: Skin - Butterfly rash
  • SLE: Affected oral sites

Oral Neoplasia and Pre-neoplasia

  • Premalignant/ potentially malignant Lesions

Premalignant Conditions

  • List of Premalignant Conditions
  • Conditions/Lesions Caused by Alcohol tobacco
  • Paterson-Kelly Syndrome: Cause and Feature

Leukoplakia | Premalignant Conditions

  • Leukoplakia diagnosis: by exclusion
  • Leukoplakia is associated with smoking
  • Leukoplakia: risk of malignant transformation - Gender
  • Leukoplakia: risk of malignant transformation- Location
  • Leukoplakia: risk of malignant transformation: Size
  • Presence of Candida Albicans increases risk of malignant transformation
  • Genetic Factors that increase the risk of malignant transformation-1
  • Genetic Factors that increase the risk of malignant transformation - 2
  • Genetic Factors that increase the risk of malignant transformation - 3

Treatment of Oral Cancer - Radiation

  • Radiation dose- how many weeks + days/week + fractions/day.

Herpes Simplex Virus-1| Viral Infections | Vesiculo-bullous Disorders

  • HSV-1: Route of infection is physical
  • HSV-1: Incubation period is 1-2 weeks
  • HSV-1: Primary infection: eruption of small vesicles
  • HSV-1: primary infection: Herpetic gingivostomatitis + affects any oral site
  • HSV-1: Primary infection: Symptoms last for 7-10 days
  • HSV-1: Virus remains latent in the trigeminal ganglion
  • Start HSV-1: Secondary infection: Vesicles rupture forming ulcers.
  • Start HSV-1: Secondary infection: Ulcers heal in 1-2 weeks

Herpes Simplex Virus-2 | Viral Infections | Vesiculo-bullous Disorders

  • HSV-2: Predilection to genital mucosa
  • HSV-2: remains latent in lambosacral ganglion
  • HSV-2: Histology: Inclusion bodies within keratinocytes.

Varicella-Zoster Virus | Viral Infections | Vesiculo-bullous Disorders

  • Primary Infection= Varicella= chicken pox.
  • VZV: Route of transmission: airborne, highly contagious.
  • VZV: incubation period: 2 weeks
  • Zoster: is an elderly disease
  • Zoster: follows a dermatomal distribution
  • Zoster: Ramsay Hunt Syndrome. (latency at cranial nerves VII and VIII)

Coxsackie Virus | Viral Infections | Vesiculo-bullous Disorders

  • manifestations are Herpangina, and “Hand, Foot, and Mouth Disease"

Hand, Foot and, Mouth disease | Viral Infections | Vesiculo-bullous Disorders

  • HFM disease: affects children
  • Hand, Foot, and mouth disease: Caused by A16 coxsackie virus
  • HFM disease: Transmission: airborne and orofecal routes

Herpangina | Viral Infections | Vesiculo-bullous Disorders

  • Herpangina: Transmitted by saliva
  • Herpangina: affects children
  • Herpangina: Endemic and seasonal (summer and early autumn)
  • Herpangina: sites: faucial pillars and soft palate.

Measles |Viral Infection | Vesiculo-bullous Disorders

  • Measles: Belongs to paramyxovirus family
  • Measles: Airborne transmission
  • Measles: Seasonal (winter and spring).
  • Measles Virus prodromal symptoms
  • Measles Virus: Incubation period
  • Measles Virus: Koplik's spots

Pemphigus | Vesiculo-bullous Disorders - Immune mediated

  • The Oral mucosa is mostly affected by 2 types of pemphigus
  • pemphigus: Reaction occurs at stratum spinosum level's desmosomes
  • pemphigus: Antibodies mainly IgG
  • Pemphigus: Antibodies become reactive to desmoglein 3
  • Pemphigus: Blister formation, no ulcers (Intra epithelial lesions)
  • Pemphigus: Possible genetic predisposition with certain MHC types..
  • Pemphigus lesions: First to show, last to go
  • Pemphigus: Positive Nikolsky's sign
  • Pemphigus: Histopathology: Tzanck cells (acantholytic cells)

Mucous Membrane Pemphigoid | Vesiculo-bullous Disorders - Immune mediated

  • M.M. Pemphigoid: separation of the epithelium from the underlying CT
  • M.M Pemphigoid: Immunoglobulins (mainly IgG)
  • MM pemphigoid: reaction to mainly Laminin 5 and BP 180 proteins.
  • MM Pemphigoid: results in oral ulcers.
  • MM pemphigoid: Mostly affects adult females
  • MM Pemphigoid: Positive Nikolsky's Sign
  • MM Pemphigoid: Outcome: Eyes: Symblepharon, which can lead to blindness.

Bullous Pemphigoid | Vesiculo-bullous Disorders - Immune mediated

  • Bullous Pemphigoid: Patients are 70-80 years
  • Bullous Pemphigoid: Antigen target is BP 230 and BP 180
  • Pemphigoid- General: Histopathology: Subepithelial clefting with no acantholysis

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